COMPENDIUM ON FUNCTIONAL MEDICINE - Flipbook - Page 583
increased permeability, inflammation, and pain in irritable bowel
syndrome. Am J Physiol Gastrointest Liver Physiol.
2012;303(7):G775-G785.
[24] Asano K, Matsushita T, Umeno J, et al. A genome-wide
association study identifies three new susceptibility loci for
ulcerative colitis in the Japanese population. Nat Genet.
2009;41(12):1325-1329.
[25] Green PH, Cellier C. Celiac disease. N Engl J Med.
2007;357(17):1731-1743.
[26] Leite G, Morales W, Weitsman S, et al. The duodenal
microbiome is altered in small intestinal bacterial overgrowth. PLoS
One. 2020;15(7):e0234906.
[27] Tlaskalová-Hogenová H, Štěpánková R, Kozáková H, et al. The
role of gut microbiota (commensal bacteria) and the mucosal
barrier in the pathogenesis of inflammatory and autoimmune
diseases and cancer: contribution of germ-free and gnotobiotic
animal models of human diseases. Cell Mol Immunol.
2011;8(2):110-120.
[28] Virili C, Centanni M. "With a little help from my friends" - The
role of microbiota in thyroid hormone metabolism and
enterohepatic recycling. Mol Cell Endocrinol. 2017;458:39-43.
[29] Scher JU, Abramson SB. The microbiome and rheumatoid
arthritis. Nat Rev Rheumatol. 2011;7(10):569-578.
[30] Mielcarz DW, Kasper LH. The gut microbiome in multiple
sclerosis. Curr Treat Options Neurol. 2015;17(4):344.
[31] Kern RC, Conley DB, Walsh W, et al. Perspectives on the
etiology of chronic rhinosinusitis: an immune barrier hypothesis. Am
J Rhinol. 2008;22(6):549-559.
[32] Hammad H, Lambrecht BN. Barrier epithelial cells and the
control of type 2 immunity. Immunity. 2015;43(1):29-40.
583
